Ahh, I was thinking about this more and I may have an explanation for my prior statement about regular albinos not having kinking. Suppose the caramel albino's mutated gene translates to a mutation in tyrosinase. The primary affect of this mutation is the obvious failure to produce melanin. On another hand, this mutation could occasionally cause the tyrosinase enzyme to react with another protein that directly impacts the skeletal development in the embryo, resulting in spinal deformities. Though, if this is the case, if you are still to assume copper content in diet has anything to do with the production of tyrosinase, then it may not be the best idea to feed high-copper diet rats to caramel albinos at all if you are attempting to avoid kinking. Now everything stated here is just speculation, but I think it might be important to take into consideration if trying an experiment as such.

Anyway, my apologies for completely derailing the thread and turning it into a cellular biology conversation with myself. I am just completely fascinated with these ideas, to the point that it's been keeping me awake tonight.